HIGHLIGHTS
SUMMARY
Strikingly, cdc5-ad and cdc5-16 were able to complement each other`s adaptation deficiency as the heterozygous cdc5-ad/cdc5-16 was adaptation proficient (Fig 5a). In contrast, the cdc5-ad-16/cdc5-ad-16 mutant was as adaptation deficient as cdc5-ad/cdc5-ad. In this model, alterations in Cdc5 levels would tip the balance of Cdc5`s fine-tuned activity, thus explaining the many results correlating adaptation efficiency with the total amount of Cdc5, as the authors see in NDD1-10A, Drts1, and strains overexpressing Cdc5, or as described previously (Hu . . .
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