Tmbim5 loss of function alters mitochondrial matrix ion homeostasis and causes a skeletal myopathy

HIGHLIGHTS

• who: Michael J Schmeisser and colleagues from the Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg-University Mainz, Mainz, Germany Department of Cellular and Molecular Medicine, KU Leuven, Laboratory of Molecular and Cellular Signaling, Leuven, Belgium Center for Translational Medicine, Lewis Katz School of Medicine at Temple University, Philadelphia, PA, USA Laboratory for Myology, Department of Human Movement Sciences, Faculty of Behavioural and Movement Sciences, Vrije Universiteit Amsterdam, Amsterdam, The Netherlands Department of Translational Research, Comprehensive Heart Failure Center (CHFC), University Clinic Wu00fcrzburg, Wu00fcrzburg, Germany Institute of Neuropathology, University Medical Center of the Johannes . . .
  

   

  

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