HIGHLIGHTS
SUMMARY
The combination of these microbial pathogens in the vagina significantly affects the host immune_response by boosting T. vaginalis-induced proinflammatory chemokine production and synergistically affecting the integrity of tight junctions between cervicovaginal epithelial_cells, which together likely contribute to a reduction in mucosal barrier function in_vivo. The interplay between dysbiotic bacteria and T. vaginalis was further confirmed by Hinderfeld and Simoes-Barbosa, who demonstrated that biofilm produced in_vitro by BV-associated bacteria is able to enhance the adhesion between protist and host cells, amplifying the parasite`s cytopathic effect. "Candidatus Mycoplasma girerdii," previously Month YYYY . . .
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